本頁只刊出中文翻譯與中文說明；英文原文請見下方原文連結。
原文連結
論文資訊
- 類型：已發表論文
- 日期：2017-01-16
摘要
演化ary theory explains why metazoan 物種 are largely protected against the negative fitness effects of 癌症s. Nevertheless, 癌症 is often observed at high incidence across a range of 物種. Although there are many challenges to quantifying 癌症 流行病學 and assessing its causes, we claim that most modern-day 癌症 in animals - and humans in particular - are due to environments deviating from central tendencies of distributions that have prevailed during 癌症 resistance 演化. Such novel environmental conditions may be natural and/or of anthropogenic origin, and may interface with 癌症 risk in numerous ways, broadly classifiable as those: increasing organism body size and/or life span, disrupting processes within the organism, and affecting germline. We argue that anthropogenic influences, in particular, explain muc
※ 此為已發表論文，全文需透過期刊付費取得