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原文連結
論文資訊
- 類型：已發表論文
- 日期：2017-05-12
摘要
Recently, an alternative theory concerning the method by which olfactory 蛋白質s are activated has garnered attention. This theory proposes that the activation of olfactory G 蛋白質-coupled receptors occurs by an inelastic electron tunneling mechanism that is mediated through the presence of an agonist with an appropriate vibrational state to accept the inelastic portion of the tunneling electron's energy. In a recent series of papers, some suggestive theoretical evidence has been offered that this theory may be applied to nonolfactory G 蛋白質-coupled receptors (GPCRs), including those associated with the central nervous system (CNS). [Chee HK, June OS (2013) Genomics Inform 11(4):282-288; Chee HK, et al. (2015) FEBS Lett 589(4):548-552; Oh SJ (2012) Genomics Inform 10(2):128-132]. Herein, we test
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