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原文連結
論文資訊
- 類型：已發表論文
- 日期：2021-04-23
摘要
Schizophrenia is marked by deficits in facial affect processing associated with abnormalities in GABAergic circuitry, deficits also found in first-degree relatives. Facial affect processing involves a distributed 網絡 of 大腦 regions including limbic regions like amygdala and visual processing areas like fusiform cortex. Pharmacological modulation of GABAergic circuitry using benzodiazepines like alprazolam can be useful for studying this facial affect processing 網絡 and associated GABAergic abnormalities in schizophrenia. Here, we use pharmacological modulation and 計算 modeling to study the contribution of GABAergic abnormalities toward emotion processing deficits in schizophrenia. Specifically, we apply principles from 網絡 control theory to model persistence energy – the control energy required
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